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Description

Protective effect of gallic acid on cyclophosphamide-induced nephrotoxicity, oxidative stress, genotoxicity, and histopathological alterations in male albino rats

The pathophysiology of Long COVID, myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS), and related dysautonomias shares a striking commonality: severe, persistent post-viral metabolic dysfunction

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Among the predicted binding targets, DRD4, 5-HT1A, 5-HT2A, Sigma1 (1), PPARG, CNR1 and CNR2 have been reported to be PD associated by previous studies 56,57,58,59,60,61,62